SARS CoV ‐2 aggravates cellular metabolism mediated complications in COVID ‐19 infection

SARS CoV ‐2 aggravates cellular metabolism mediated complications in COVID ‐19 infection.

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Item Type: Article
Status: Published
Official URL: https://doi.org/10.1111/dth.13871
Journal or Publication Title: Dermatologic Therapy
Date: 2020
Divisions: UTS Centre for Inflammation
Depositing User: General Admin
Identification Number: 10.1111/dth.13871
ISSN: 1396-0296
Date Deposited: 03 Jan 2021 23:51
Abstract:

Severe acute respiratory syndrome coronavirus‐2 (SARS‐CoV‐2), the primary causative organism in corona virus disease‐19 (COVID‐19) infections, is a novel member of the human coronavirus family which was first identified in Wuhan, China, towards the end of 2019. This letter reveals new vital missing links in our current understanding of the mechanisms that lead to cell death triggered by ferroptotic stress in COVID‐19 infection. It further reveal the importance of homocysteine mediated trans‐sulfuration pathway in COVID‐19 infection. Hence, Vitamin B6, folic acid, and Vitamin B12 should be incorporated in the treatment regimen for SARS CoV‐2 infections to suppress complications, as the virus mediates altered host cell metabolism.

Creators:
Creators
Email
Singh, Yogendra
UNSPECIFIED
Gupta, Gaurav
UNSPECIFIED
Kazmi, Imran
UNSPECIFIED
Al‐Abbasi, Fahad A.
UNSPECIFIED
Negi, Poonam
UNSPECIFIED
Chellappan, Dinesh Kumar
UNSPECIFIED
Dua, Kamal
UNSPECIFIED
Last Modified: 03 Jan 2021 23:51
URI: https://eprints.centenary.org.au/id/eprint/836

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